FOXP3 Is an X-Linked Breast Cancer Suppressor Gene and an Important Repressor of the HER-2/ErbB2 Oncogene

نویسندگان

  • Tao Zuo
  • Lizhong Wang
  • Carl Morrison
  • Xing Chang
  • Huiming Zhang
  • Yan Liu
  • Yin Wang
  • Xingluo Liu
  • Michael W.Y. Chan
  • Jin-Qing Liu
  • Richard Love
  • Virginia Godfrey
  • Rulong Shen
  • Tim H-M. Huang
  • Tianyu Yang
  • Pan Zheng
  • Yang Liu
چکیده

The X-linked Foxp3 is a member of the forkhead/winged helix transcription factor family. Germline mutations cause lethal autoimmune diseases in males. Serendipitously, we observed that female mice heterozygous for the "scurfin" mutation of the Foxp3 gene (Foxp3(sf/+)) developed cancer at a high rate. The majority of the cancers were mammary carcinomas in which the wild-type Foxp3 allele was inactivated and HER-2/ErbB2 was overexpressed. Foxp3 bound and repressed the HER-2/ErbB2 promoter. Deletion, functionally significant somatic mutations, and downregulation of the FOXP3 gene were commonly found in human breast cancer samples and correlated significantly with HER-2/ErbB2 overexpression, regardless of the status of HER-2 amplification. Our data demonstrate that FOXP3 is an X-linked breast cancer suppressor gene and an important regulator of the HER-2/ErbB2 oncogene.

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عنوان ژورنال:
  • Cell

دوره 129  شماره 

صفحات  -

تاریخ انتشار 2007